Bottled Water: the biggest marketing con of them all.

..and many of them have fluoride added!

Is Fluoride in our water really working?

..and if it isn’t, why are we forced to drink fluoridated water?

In a study that the authors describe as lending credence to the idiom, “by the skin of your teeth,” scientists are reporting that the protective shield fluoride forms on teeth is up to 100 times thinner than previously believed. It raises questions about how this renowned cavity-fighter really works and could lead to better ways of protecting teeth from decay, the scientists suggest. Their study appears in ACS’s journal Langmuir.

Frank M-ller and colleagues point out that tooth decay is a major public health problem worldwide. In the United States alone, consumers spend more than $50 billion each year on the treatment of cavities. The fluoride in some toothpaste, mouthwash and municipal drinking water is one of the most effective ways to prevent decay. Scientists long have known that fluoride makes enamel – the hard white substance covering the surface of teeth – more resistant to decay. Some thought that fluoride simply changed the main mineral in enamel, hydroxyapatite, into a more-decay resistant material called fluorapatite.

The new research found that the fluorapatite layer formed in this way is only 6 nanometers thick. It would take almost 10,000 such layers to span the width of a human hair. That’s at least 10 times thinner than previous studies indicated. The scientists question whether a layer so thin, which is quickly worn away by ordinary chewing, really can shield teeth from decay, or whether fluoride has some other unrecognized effect on tooth enamel. They are launching a new study in search of an answer.

Source: American Chemical Society

When a doctor makes a mistake. A courageous man.

The Dalai Lama talks about health

Hydrogen-Rich Water cited for use in reducing the effects of radiation from Fukushima

It doesn’t take long for scientists to see the value of technologies that were ignored until the magnitude of the problem gets to ‘overload’.

It’s fair to say that the Fukushima meltdown in Japan is such an ‘overload’ situation, and this is why scientists at  the Institute of Radiation Emergency Medicine, in Hirosaki, Japan, are suggesting that hydrogen-rich water similar to the water produced by our water ionizers be considered as a method of countering the runaway free radical activity created by radiation exposure. For me it’s validation of what we’ve believed for a decade – that water rich in hydrogen is a powerful antioxidant, possibly better than many antioxidant supplements and definitely better than most antioxidant-rich foods.

 

For readers interested in learning more, the study linked above linked and refers to this study and this one.

Metabolic Syndrome and Alkaline Water

As usual, when reporting on the growing volume of scientific studies of alkaline water, we need to tell you that if you scroll down to the article below you agree that you understand we are NOT offering any form of Therapeutic advice, nor are we saying or inferring that the consumption of alkaline water may have a beneficial effect upon your on health. We are bound by law to advise you that before you make any change to your current heath regimen you should seek the advice of a registered health professional.

Scroll down to read…

Metabolic Syndrome and the Effects of Alkaline  Water: A study.

Research team: Cidália Pereira, Rosário Monteiro, Alejandro Santos, Maria João Martins

From: ( mmartins@med.up.pt)

Adapted from the publication on 
Nutrição. In: Guias de Saúde. Volume 7. Vila do Conde: QuidNovi

Metabolic Syndrome.

The Metabolic Syndrome (MetSyn; also called Syndrome X or Insulin Resistance Syndrome) is a cluster of metabolic abnormalities that increase the risk of developing atherosclerotic cardiovascular disease and type 2 diabetes mellitus (or is associated with type 2 diabetes mellitus).

Individual components that define MetSyn include atherogenic dyslipidemia (alteration of blood lipid profile favouring atherosclerosis development and being characterized by high fasting blood triglycerides and low fasting blood HDL-cholesterol), elevated fasting blood glucose and (or) insulin resistance (more insulin is need to control/regulate blood glucose levels), elevated blood pressure, abdominal obesity and, most recently recognized, a pro-inflammatory and prothrombotic state [a state favouring inflammation and thrombus (blood clot) formation] (Zimmet et al 2005; Johnson et al 2006; Grundy et al 2006;  Feldeisen et al 2007; Alberti et al 2009; Simmons et al 2010; Wree et al 2011).

The increasing number of individuals with MetSyn, in the past 10-15 years, has been associated with several different factors. Although the exact aetiology of the MetSyn still remains unclear, it is known to involve complex interactions between genetic, metabolic and environmental factors. Among environmental factors, diet and physical activity are of central importance in the prevention and treatment of this condition. Some minerals, like calcium, magnesium and potassium, generally deficient in MetSyn-inducing diets, due to a low ingestion of milk, dairy products, fruit, vegetables, whole grains, beans and nuts, like almonds and walnuts, have been proposed protective against the MetSyn (Feldeisen et al 2007).

Minerals and the Metabolic Syndrome.

The high intake of sodium on one hand and the low intakes of potassium, calcium and magnesium on the other hand, produce and maintain elevated blood pressure in a big proportion of the population. Conversely, decreased intake of sodium alone, and increased intakes of potassium, calcium and magnesium, each alone, decrease elevated blood pressure. A combination of all these factors, that is, decrease of sodium, and increase of potassium, calcium and magnesium intakes, which are characteristic of the so-called Dietary Approaches to Stop Hypertension (DASH) diets, has an excellent blood pressure lowering effect (Van Leer et al 1995; Whelton et al 1997; Karppanen et al 2005; Geleijnse et al 2005; van Meijl et al 2008).
Research has indicated that low intake of magnesium, low blood magnesium concentrations and/or low intracellular magnesium levels may lead to and are associated with elevated blood pressure, MetSyn, insulin resistance, and/or type 2 diabetes mellitus (Song et al 2004; He et al 2006; Volpe et al 2008; Wells 2008). Experimental and clinical studies suggest that magnesium intake may decrease blood triglyceride and increase HDL-cholesterol levels (He et al 2006). Both individuals who did not have type 2 diabetes mellitus, but with insulin resistance and hypomagnesemia (low blood magnesium level), and individuals with type 2 diabetes mellitus, with hypomagnesemia, showed improved insulin sensitivity and, for type 2 diabetic individuals, improved metabolic control (lower fasting blood glucose and lower glycated haemoglobin levels), after oral magnesium supplementation (Song et al 2004; Volpe et al 2008; Wells 2008). A strong inverse relationship between magnesium levels in serum and the presence of MetSyn has been reported, in a population of overweight or obese individuals (mean age around 66 years), in which serum magnesium levels decreased as the number of components of MetSyn increased (Evangelopoulos et al 2008).

Epidemiological studies have suggested protective effects of dairy product consumption on MetSyn development. Additionally, it has been published that calcium supplements improve the serum lipoprotein profile, particularly by decreasing serum total and LDL-cholesterol concentrations (van Meijl et al 2008). In overweight or obese women (mean age 43 years), who were very low-calcium consumers, decreases in body weight, fat mass and spontaneous dietary lipid intake have been associated with calcium plus vitamin D supplementation, for 15 weeks (Major et al 2009). Based on the Korean National Health and Nutrition Examination Survey (2001 and 2005) calcium intake is inversely associated with the risk of having MetSyn in postmenopausal women (Cho et al 2009).

Drinking water and its mineral content.

Several investigations evaluated the relationship between hardness of drinking water, or its content in magnesium and calcium, and the risk for cardiovascular disease or stroke. Results support the hypothesis that a low intake of magnesium in drinking water may increase the risk of dying from, and possibly developing, cardiovascular disease or stroke (Monarca et al 2006; Rylander 2008). An additional parameter to take into account is the acidity of the water (there is considerable evidence that acid-base conditions in the body influence the mineral homeostasis and it is known that acid load influences the reabsorption of calcium and magnesium in renal tubuli). It has been suggested that the health effects related to drinking water found in some studies may have been caused by an increased urinary excretion of minerals induced by acid conditions in the body and that drinking water should contain sufficient amounts of hydrogen carbonate to prevent this effect (Rylander et al 2006; Rylander 2008).
Natural mineral waters represent a substantial alkaline load and may influence mineral homeostasis in our body (Rylander 2008). Several papers in the literature point to calcium- and (or) magnesium-rich natural mineral waters as good sources of these ions (in which they are highly bioavailable), contributing to achieve their daily recommended intakes (Bohmer et al 2000; Sabatier et al 2002; Bacciottini et al 2004; Kiss et al 2004; Heaney 2006; Karagülle et al 2006). 

It is interesting to mention that, besides the influence on MetSyn components (see below), the mineral content of natural waters may have other preventive/beneficial effects.  It has been reported that in a Hungarian city the occurrence of preeclampsia varied pari passu with the magnesium content of the drinking water in different parts of the city (Melles et al 1992). In a different study, the consumption of 1L/day of a high calcium natural mineral water (supplement of 596 mg of calcium), for 6 months, reduced serum parathyroid hormone and indices of bone turnover in postmenopausal women with a low calcium intake (Meunier et al 2005).

Natural mineral waters and Metabolic Syndrome components.

Within the scope of beneficial effects in cardiovascular disease and MetSyn prevention, there are several publications showing that the ingestion of mineral waters with sodium bicarbonate is beneficial in lowering cardiovascular risk factors, including blood pressure (Luft et al 1990; Schorr et al 1996; Capurso et al 1999; Rylander et al 2004; Schoppen et al 2004; Almeida et al 2010a,b; Pérez-Granados et al 2010).

The consumption of 3L/day of a NaHCO3-containing mineral water, for 7 days, decreased systolic blood pressure, in mildly hypertensive men (Luft et al 1990) and the consumption of 1.5L/day of a sodium bicarbonate-rich mineral water, for 4 weeks, decreased mean arterial blood pressure, in elderly normotensive subjects (aged 60-72 years) (Schorr et al 1996). The daily ingestion of 0.5 mL of a portuguese natural mineral water rich in bicarbonate and sodium, Água das Pedras® (and with a higher content in magnesium, calcium and potassium than tap water from Porto city area, where the study took place), for 7 weeks, had no effect on blood pressure, in normotensive adults (aged 24-53 years) (Santos et al 2010). Also, administration of this natural mineral-rich water in an animal model of MetSyn did not increase blood pressure and improved some metabolic parameters (like plasma insulin and triglycerides levels) (Almeida 2010a,b).

Ingestion of a natural mineral water rich in calcium, bicarbonate and magnesium, as well as in sulphate, reduced blood pressure (systolic and diastolic) after 2 weeks (this reduction was kept until the 4 weeks of treatment) in individuals (aged 45 – 64 years) with borderline hypertension and with low urinary excretion of magnesium and calcium (Rylander et al 2004). In moderately hypercholesterolemic young adults (aged 18 – 40 years), ingestion of a bicarbonated natural mineral water (also rich in sodium, chloride and potassium; 1L/day), for 8 weeks, reduced systolic blood pressure (this alteration was observed after 4-weeks consumption, without significant differences between weeks 4 and 8), fasting serum levels of apolipoprotein B, total cholesterol and LDL-cholesterol as well as the ratios [(total cholesterol)/(HDL-cholesterol)] and [(LDL-cholesterol)/(HDL-cholesterol)] (Pérez-Granados et al 2010). In postmenopausal women, ingestion of the previous natural mineral-rich water (1L/day), for 2 months, increased fasting serum levels of HDL-cholesterol and reduced fasting serum levels of two markers of endothelial dysfunction, glucose, total cholesterol and LDL-cholesterol as well as the ratios [(total cholesterol)/(HDL-cholesterol)] and [(LDL-cholesterol)/(HDL-cholesterol)] (Schoppen et al 2004).

Conclusion.

Presently, with the increase in MetSyn and type 2 diabetes mellitus, associated with a high consumption of calorie-rich and micronutrient-poor foods, ingestion of natural mineral-rich waters may be beneficial. This effect may be even greater if ingestion of sweetened beverages is replaced by natural mineral-rich waters (Schulze et al 2004; Vartanian et al 2007; Feldeisen et al 2007).

References.
. Alberti KG et al. Circulation. 2009; 120(16): 1640-5.
. Almeida C et al. Chronic ingestion of a hypersaline sodium-rich carbonated natural mineral water on an animal model of the metabolic syndrome – effects on blood pressure and plasma metabolic profile. Press Therm Climat. 2010a; 147: 110-1.
. Almeida C et al. Effects of mineral supplementation on a wide spectrum of Metabolic Syndrome features. Study performed on a fructose-fed animal model. Public Health Nutr. 2010b; 13: 234.
. Bacciottini L et al. Calcium bioavailability from a calcium-rich mineral water, with some observations on method. J Clin Gastroenterol. 2004; 38(9): 761-6.
. Bohmer H et al. Calcium supplementation with calcium-rich mineral waters: a systematic review and meta-analysis of its bioavailability. Osteoporos Int. 2000; 11(11): 938-43.
. Capurso A et al. Increased bile acid excretion and reduction of serum cholesterol after crenotherapy with salt-rich mineral water. Aging (Milano). 1999 Aug;11(4):273-6.
. Cho GJ et al. Calcium intake is inversely associated with metabolic syndrome in postmenopausal women: Korea National Health and Nutrition Survey, 2001 and 2005. Menopause. 2009; 16(5): 992-7.
. Evangelopoulos AA et al. An inverse relationship between cumulating components of the metabolic syndrome and serum magnesium levels. Nutr Res. 2008; 28(10): 659-63.
. Feldeisen SE et al. Nutritional strategies in the prevention and treatment of metabolic syndrome. Appl Physiol Nutr Metab. 2007; 32(1): 46-60.
. Geleijnse JM et al. Impact of dietary and lifestyle factors on the prevalence of hypertension in Western populations. J Hum Hypertens. 2005 Dec;19 Suppl 3:S1-4.
. Grundy SM et al. Diagnosis and management of the metabolic syndrome: an American Heart Association/National Heart, Lung, and Blood Institute scientific statement. Curr Opin Cardiol. 2006 Jan;21(1):1-6.
. Heaney RP. Absorbability and utility of calcium in mineral waters. Am J Clin Nutr. 2006; 84(2): 371-4.
. Johnson LW et al. The metabolic syndrome: concepts and controversy. Mayo Clin Proc. 2006; 81(12): 1615-20.
. Karagülle O et al. Magnesium absorption from mineral waters of different magnesium content in healthy subjects. Forsch Komplementmed. 2006; 13(1): 9-14.
. Karppanen H et al. Why and how to implement sodium, potassium, calcium, and magnesium changes in food items and diets? J Hum Hypertens. 2005; 19 Suppl 3: S10-9.
. Kiss SA et al. Absorption and effect of the magnesium content of a mineral water in the human body. J Am Coll Nutr. 2004; 23(6): 758S-62S.
. Luft FC et al. Sodium bicarbonate and sodium chloride: effects on blood pressure and electrolyte homeostasis in normal and hypertensive man. J Hypertens. 1990; 8(7): 663-70.
. Major GC et al. Calcium plus vitamin D supplementation and fat mass loss in female very low-calcium consumers: potential link with a calcium-specific appetite control. Br J Nutr. 2009; 101(5): 659-63.
. Melles Z et al. Influence of the magnesium content of drinking water and of magnesium therapy on the occurrence of preeclampsia. Magnes Res. 1992 Dec;5(4):277-9.
. Meunier et al. Consumption of a high calcium mineral water lowers biochemical indices of bone remodeling in postmenopausal women with low calcium intake. Osteoporos Int. 2005 Oct;16(10):1203-9.
. Monarca S et al. Review of epidemiological studies on drinking water hardness and cardiovascular diseases. Eur J Cardiovasc Prev Rehabil. 2006; 13(4): 495-506.
. Pérez-Granados AM et al. Reduction in  cardiovascular risk by sodium-bicarbonated mineral water in moderately hypercholesterolemic young adults. J Nutr Biochem. 2010; 21(10): 948-53.
. Rylander R et al. Acid-base status affects renal magnesium losses in healthy, elderly persons. J Nutr. 2006;136(9):2374-7.
. Rylander R et al. Mineral water intake reduces blood pressure among subjects with low urinary magnesium and calcium levels. BMC Public Health. 2004; 4: 56.
. Rylander R. Drinking water constituents and disease. J Nutr. 2008; 138(2): 423S-425S.
. Sabatier M et al. Meal effect on magnesium bioavailability from mineral water in healthy women. Am J Clin Nutr. 2002; 75(1): 65-71.
. Santos A et al. Sodium-rich carbonated natural mineral water ingestion and blood pressure. Rev Port Cardiol. 2010; 29(2): 159-72.
. Schoppen S et al. A sodium-rich carbonated mineral water reduces cardiovascular risk in postmenopausal women. J Nutr. 2004; 134(5): 1058-63.
. Schorr U et al. Effect of sodium chloride- and sodium bicarbonate-rich mineral water on blood pressure and metabolic parameters in elderly normotensive individuals: a randomized double-blind crossover trial. J Hypertens. 1996; 14(1): 131-5.
. Schulze MB et al. Sugar-sweetened beverages, weight gain, and incidence of type 2 diabetes in young and middle-aged women. JAMA. 2004; 292(8): 927-34.
. Simmons RK et al. The metabolic syndrome: useful concept or clinical tool? Report of a WHO Expert Consultation. Diabetologia. 2010; 53(4): 600-5.
. Song Y et al. Dietary magnesium intake in relation to plasma insulin levels and risk of type 2 diabetes in women. Diabetes Care. 2004; 27(1): 59-65.
. Van Leer EM et al. Dietary calcium, potassium, magnesium and blood pressure in the Netherlands. Int J Epidemiol. 1995; 24(6): 1117-23.
. van Meijl LE et al. Dairy product consumption and the metabolic syndrome. Nutr Res Rev. 2008; 21(2): 148-57.
. Vartanian LR et al. Effects of soft drink consumption on nutrition and health: a systematic review and meta-analysis. Am J Public Health. 2007; 97(4): 667-75. 
. Volpe SL. Magnesium, the metabolic syndrome, insulin resistance, and type 2 diabetes mellitus. Crit Rev Food Sci Nutr. 2008; 48(3): 293-300.
. Wells IC. Evidence that the etiology of the syndrome containing type 2 diabetes mellitus results from abnormal magnesium metabolism. Can J Physiol Pharmacol. 2008; 86(1-2): 16-24.
. Whelton PK et al. Effects of oral potassium on blood pressure. Meta-analysis of randomized controlled clinical trials. JAMA. 1997; 277(20): 1624-32.
. Wree A et al. Obesity affects the liver – the link between adipocytes and hepatocytes. Digestion. 2011; 83(1-2): 124-33.
. Zimmet P et al. The metabolic syndrome: a global public health problem and a new definition. J Atheroscler Thromb. 2005; 12(6): 295-300.

The Ultimate Insult to a visitor: being told ‘you smell’ by a machine.

We are just getting used to our in-laws’ Roomba robot vacuum cleaner. We go to visit and the Roomba is puttering away as we sit and talk.. very kool for a gadget freak like me. But now there’s a new device that will sneak into our homes innocuously (like a Roomba).. and perhaps cause major social disturbance. It’s a mobile robot air freshener..  seen here firmly encased in its own glass cage.. and no wonder!Just think.. your ‘Rydis’ ( that’s its name -disturbingly similar to Tardis..) is happily sniffing out pockets of relatively poor quality air around the home. It seems to spend a long time loitering at the door of the loo for some reason.. until your mother-in-law of er.. advanced years comes to stay for her annual checkup on how her beloved daughter is being treated but tha no-good son-in-law.

YOU know she smells – many old people do – but you’d never say anything. It’s just part of the price you are willing to pay for marital continuity. Suddenly mother-in-law has a new friend. Rydis is tracking her. Rydis is sitting in front of her s she watches Oprah. Rydis turns its fans on full blast every time it locates her. It even follows her to her bedroom!

And you have to explain to her why. No thanks.

Inflammation and Heart Attack

Cholesterol, cholesterol, cholesterol.

It’s certainly the battlecry of the general practitioner and the word heard most frequently around the subject of heart disease. In fact it’s heard so often that all the other equally important factors seem to be in the shade of the almighty ‘C” word. I’m not going to talk about whether or not cholesterol is the problem we are told it is.. that’d take too long and I might get over-excited. But I would like to look at the other factors.. and a common symptom of all of them. What about tobacco usage, psychosocial stress, activity level, or  genetic predisposition? These are all heart disease factors. But exactly how do they contribute to heart disease?

Ask most doctors and they’ll agree that beyond all of them, inflammation is the root cause of a heart attack. All of the factors create inflammation, which in turn, creates the conditions that may precipitate a heart attack.

Fags

A simple explanation of the relationship between smoking and inflammation is to show what happens when a smoker stops smoking, as in this Reuters article, which describes a study by Dr. Christine N. Metz and her team. Very simply, researchers  watched inflammation markers during a smokers’ ‘quit’ program. And yes, inflammation levels dropped quickly. If you need further evidence of the effect of inhaling hot tar-filled smoke into your fragile internal environment, this study shows the direct link between smoking, inflammation and heart disease. The study even slates passve smoking, saying :

“Passive smoke itself, is a volatile mixture of numerous toxins, chemicals and carcinogens, that interact with in vivo mechanisms and induce vascular damage, including endothelium inflammation, atherosclerosis development, lipid peroxidisation, alterations in cytokines and acute phase proteins (such as CRP), as well as platelet aggravation.”

Another PubMed study links smoking directly to inflammation and atherosclerotic plaque. In other words, big chunks of plaque that can break off with a good cough and block an artery – with fairly obvious results. This study showed the possibility of inflammation AND plaque combining to create an ‘event’.

Stress and Inflammation

It’s now shown that stress of all kinds creates an inflammation response. It’s interesting because we have learned to ‘control’ stress in daily life, but I wonder just what levels we are faced with compared to our ancestors. The stress of climate change, of financial system collapse, of job insecurity don’t go away. They remain an enduring part of what we have to manage every day. This report demonstrates that it is the repetition of stress that causes inflammation.. but it actually describes an example of chronic inflammatory response as atherosclerosis.

There are even studies that link anger and cynicism to inflammation.

Summarizing, this paper links stress events via inflammation to heart attacks. “The argument is made that humans reacting to stressors, which are not life-threatening but are “perceived” as such, mount similar stress/inflammatory responses in the arteries, and which, if repetitive or chronic, may culminate in atherosclerosis.” In reading up on this subject you’ll come across another term ‘Oxidative stress’. Oxidative stress is what happens as a result of inflammation. As Sang Whang said in  ’Reverse Aging‘, inflammation and acids go hand in hand. Oxidation of tissue is caused by the acids that gather in inflamed sites. The stress load of oxidation travels, and as acid, it oxidises our good cholesterol, our LDL. The problem is.. oxidised LDL becomes plaque.

If we had one study describing the relationship of inflammation to heart attack we may be able to dismiss it, but when we see so many, all saying the same thing.. that inflammation and heart disease are inextricably linked, it’s probably time to take notice.

I’m not qualified to advise on heart disease. I’m a layman like most of my readers, and you should consult your medical practitioner before acting upon any of my crazy theories..

However it certainly seems to me that the circuitbreaker between inflammation and oxidative stress may just be something as simple as a constant program of maintaining a healthy pH balance. Given that water is one of the body’s first line tools for draining acids from sites of inflammation, it seems reasonable to me that alkaline microclustered water just may assist. As my readers know, I am not permitted by law to make any therapeutic claims, nor am I permitted to relate the many stories of our clients.

So I guess you’ll just have to research the subject yourself, won’t you?

Oh! Someone talked to me!

Although all of my posts here go automatically over to our Facebook page, few people make any comments.. which makes me a bit sad. I would like more people to talk to me, even if it’s to disagree! Here’s the link to our Facebook page.

If you ‘Like’ it, you’ll get automatic updates on your Facebook page.

If you can’t be bothered scrolling through all the posts here, you can do it far quicker on Facebook.

If you want to do it even faster, yet keep up to date, here’s our link to Twitter that also updates with every post here. Just click on ‘Follow’ and you’ll be notified of all my updates.

Cholesterol, Saturated Fats, Heart Disease.. the tide is turning… and an opportunity.

Another excellent little video summing up to massive con-job that cholesterol and saturated fats is!

Which brings me to the question… as you know, I have 6 tablespoons of coconut oil every day to keep me from slipping back into my incipient Alzheimers’. I’m wondering if there are others in readerland who might be interested in a group where members participate in bulk purchase of coco oil. I have the green light from the world’s best producers of organic coconut oil, and they offer 4l pails, which are far more economical than the 1L bottles. If I hear from you, I’ll consider it, but if not, that’s OK too because I already get my ‘good oil’ wholesale.

Oh yes, here’s the video that people are raving about; Cassie and I discussing my Alzheimers’.
It’sgone viral on YouTube

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